Atherosclerosis


What is atherosclerosis?

Atherosclerosis or arteriosclerosis refers to a buildup of plaques in the arteries and thus their hardening and narrowing.

Atherosclerosis word origin: from the Greek atheros = gruel – a soft, cooked cereal, which refers to a plaque + sclerosis = hardening [3]

Arterial Plaque (Atheroma) Composition and Development

Arterial plaque is an abnormal deposition of fatty acids, cholesterol, calcium, inflammatory cells and connective tissue on the inner side of the large and medium-sized arteries [53]. Plaques can start to develop in childhood; most older people probably have some plaques but these obstruct the blood flow only when they occupy 50-70% of the artery diameter [2].

A plaque develops on the inner surface (intima) of the arteries that are damaged due to [2,53]:

  • Nicotine from tobacco smoke
  • High homocysteine levels (sometimes genetic or due to folate deficiency)
  • High angiotensin II levels (increased in high blood pressure)
  • Hyperglycemia > oxidative stress > free radicals > small oxidized LDL cholesterol particles
  • Systemic infection or inflammation
  • Turbulent blood flow

The above factors can inactivate nitric oxide, which normally dilates the arteries and decreases inflammation in them [53].

A plaque can be stable or unstable. An unstable plaque, even if it appears small on angiogram, can rupture and a blood clot (thrombus) can develop on the top of it and additionally obstruct the artery [53]. A part of the thrombus (embolus) can detach and lodge further in the artery where its diameter is smaller and completely blocks it, which can cause, for example, heart attack.

Causes and Risk Factors

The exact cause of atherosclerosis is not known but involves a damage and inflammation of the inner arterial layer (intima).

Main risk factors [2,5]:

  • Genetic factors (family history of premature coronary heart disease or stroke, familial hypercholesterolemia, male sex)
  • Older age
  • Cigarette smoking
  • High levels of total and LDL cholesterol and triglycerides and low levels of HDL cholesterol
  • High blood glucose levels (hyperglycemia in poorly controlled diabetes mellitus type 1 or type 2 [insulin resistance])
  • High blood pressure (hypertension)

Other possible risk factors [5,53]:

  • Metabolic syndrome (a combination of abdominal obesity, high blood pressure, high glucose and triglyceride levels and low HDL cholesterol levels); the main causes are probably lack of physical inactivity and insulin resistance
  • Excessive alcohol drinking (moderate alcohol drinking with <2 drinks/day for men and <1 drink/day for women does not increase the risk)
  • Lack of antioxidants (vitamin A, C, E, selenium, flavonoids) in the diet
  • High levels of small oxidized LDL particles, fibrinogen and lipoprotein(a), low levels of alphalipoprotein
  • Chronic inflammatory disease, such as systemic lupus erythematosus (SLE), rheumatoid arthritis or systemic sclerosis [1]
  • Chronic kidney disease
  • Infection with Chlamydophila pneumoniae, Helicobacter pylori or citomegalovirus (CMV)
  • Psychological stress, depression
  • Living near a busy road (long-term exposure to traffic-related dust and night-time traffic noise) [54]

Symptoms, Signs and Complications

Mild atherosclerosis may not cause any symptoms but severe atherosclerosis can result in [2]:

  • Narrowing of the coronary arteries (coronary heart disease) with chest pain (angina pectoris) triggered by exercise or stress; or, eventually, heart attack (myocardial infarct), which can be deadly
  • Narrowing of the carotid arteries in the neck or cerebral arteries in the brain, which can result in transient ischemic attack (TIA) or stroke (cerebral infarct) with a sudden loss of consciousness, muscle paralysis and loss of sensation on one side of the body or death
  • Narrowing of the arteries in the legs (peripheral artery disease or arteriosclerosis obliterans) withe pain, tingling and numbness triggered by walking (intermittent claudication)
  • Narrowing of the arteries in the pelvis with pelvic pain and erectile dysfunction
  • Renal artery stenosis, which can result in chronic kidney disease with nausea, increased urination and leg swelling
  • Narrowing and hardening of the abdominal aorta, which can cause abdominal pain (only in late stage)
  • Narrowing of the intestinal (mesenteric) arteries, which can cause pain after meals, black stools, diarrhea and weight loss

Atherosclerosis in early stages usually does not cause any signs. In developed atherosclerosis, a doctor can detect weak pulse in the posterior tibial artery behind the inner ankles, radial artery in the wrists or carotid artery in the neck, and, sometimes, bluish discoloration (cyanosis) of the lower legs and leg ulcers.

Atherosclerosis is more common in men than in women and it causes symptoms in mean earlier (typically after 45 years of age) than in women (usually after 55 years of age) [2]. Estrogen hormones seem to be somewhat protective against atherosclerosis, so this is why women often develop it after menopause, when the estrogen levels fall [53].

Diagnosis

Most people with atherosclerosis who have no symptoms are not aware of the condition. Sometimes, a doctor accidentally detects atherosclerosis during investigations for other conditions, such as chest ultrasound or computed tomography (CT) of the brain.

Atherosclerosis can be detected by the following investigations:

  • CT angiography as the initial investigation
  • Coronary angiography or cardiac catheterization (an X-ray image of the coronary arteries after injecting a contrast substance into them)
  • Electron-beam computed tomography (EBCT)
  • Nuclear perfusion imaging with single-photon emission computed tomography (SPECT)
  • Positron emission tomography (PET)
  • Carotid artery intima-media thickness (CIMT) measured by ultrasound
  • Intravascular ultrasound (IVUS)
  • Magnetic resonance imaging (MRI)

Treatment

Atherosclerosis cannot be cured but it can be treated.

Drugs

The following drugs can reduce the risk factors for atherosclerosis [2,53]:

  • To lower high LDL cholesterol: statins (atorvastatin, lovastatin, simvastatin) [4] and bile acid sequestrans (cholestyramine, colestipol)
  • To raise low HDL cholesterol: vitamin B3 or niacin (insufficient evidence of additional effectiveness when used with statins) [55,56], fibrates (fenofibrate)
  • To reduce blood clotting: aspirin [4] or clopidogrel
  • To lower high blood pressure: ACE inhibitors [57], beta-blockers [58]

Smoking cessation [61], weight loss (in overweight individuals with high blood pressure) [62] and aggressive treatment of high LDL cholesterol and blood pressure [59,60] can–at least partly–reverse atherosclerosis.

Surgery

  • In coronary heart disease: coronary artery angioplasty or coronary artery bypass grafting (CABG)
  • In atherosclerosis of the carotid artery: carotid endarterectomy (removal of a plaque from the artery)
  • In atherosclerotic ileal arteries or arteries in the lower legs: by pass surgery

Prevention

If you have a strong family history of coronary heart disease or stroke, you may be not able to completely prevent atherosclerosis but you may slow down its development.

Diet

Vegetarian [33,34,35] and Mediterranean diet (whole grains, fruits vegetables, olive oil, nuts, seeds, fish, wine) [36,37,38,39,40] have been associated with the lower risk of atherosclerosis than other diets.

There is SOME EVIDENCE that replacing some of saturated fats (processed meat, beef, pork and lamb) with polyunsaturated fats (fatty fish) in the diet can reduce the risk of coronary heart disease [63,64,65].

Regular consumption–at least twice weekly–of fatty fish (mackerel, salmon, sardines, halibut, trout), which are high in omega-3 polyunsaturated fatty acids EPA and DHA have been associated with lower risk of coronary artery disease [35,48,49]. However, there is INSUFFICIENT EVIDENCE that fish oil supplements would help to prevent coronary heart disease or atherosclerosis in general [28,29,30,48,49].

In some people with high blood pressure and high sodium intake, reducing sodium intake can lower blood pressure and thus decrease the risk of coronary heart disease [50,51].

High consumption of trans fat from foods like French fries, cheeseburgers, chicken nuggets, commercially baked biscuits, crackers, doughnuts and pies, hard margarines, vegetable shortening, other foods with added “partially hydrogenated oil” or Vanaspati ghee have been associated with increased risk of atherosclerosis [43,44,45,46,47].

High consumption of processed meat (bacon, bologna, corned beef, ham, hamburgers, hot dogs, deli or luncheon meats, salami and sausages), which contain preservatives, such as salt, nitrites, nitrates and nitrosamine, but not unprocessed red meat (beef, pork and lamb), has been associated (not necessary cause-effect related) with higher incidence of coronary heart disease [52].

Supplements and Herbs

There is INSUFFICIENT EVIDENCE about the preventative effect of the following supplements on atherosclerosis:

  • Minerals: calcium [20], chromium [28], iron [28], magnesium [19,28], potassium [21]selenium [28]
  • Vitamins: beta carotene [6], vitamin A, C [28] or E [6,13,28], which are all antioxidants, vitamin B6, B12 or folate [14,28], vitamin D or K [28], choline [15]
  • Phytonutrients: lycopene [28], flavanols in green or black tea [9] chocolate and cocoa [24,25,28], flavonoids, including resveratrol (fruits, vegetables, red wine) [10,28], soy isoflavones [12], phytosterols [17,18]
  • Herbs: Ayurvedic medicine (curcumin from turmeric [31,41], ginger [32], mukul myrrh tree [28]), Chinese herbal medicine (Danshen, ginkgo biloba, ginseng, horse chestnut, lingzhi, red yeast rice extract, rosemary extract, Tong-xin-luo [7]) [27,28], garlic [16,28]
  • Other: coenzyme Q10 [8,28], alpha-linolenic acid (flaxseed oil) [11], monounsaturated fatty acids from olive oil or nuts [22,23], L-carnitine [28]

Atherosclerosis Prevention and Reversal

To reverse or at least slow down the development of atherosclerosis, try [59,60,61,62]:

  1. Maintain healthy body weight.
  2. Maintain normal blood pressure.
  3. Maintain normal blood cholesterol, triglyceride and glucose levels.
  4. Eat what helps you achieve the above goals.
  5. Be physically active (aerobic exercise, such as walking or running seems to be most protective)
  6. Do not smoke.


References
  • Au K et al, 2011, Atherosclerosis in systemic sclerosis: a systematic review and meta-analysis  PubMed
  • Boudy FB, Noncoronary atherosclerosis  Emedicine
  • Ladich ER, Atherosclerosis Pathology  Emedicine
  • Karmali KN et al, 2016, Drugs for Primary Prevention of Atherosclerotic Cardiovascular Disease, An Overview of Systematic Reviews  JAMA Cardiology
  • Boudi FB, Coronary artery atherosclerosis  Emedicine
  • Lönn ME et al, 2012, Actions of “antioxidants” in the protection against atherosclerosis  PubMed
  • Mao C et al, 2015, Tong-xin-luo capsule for patients with coronary heart disease after percutaneous coronary intervention  Cochrane
  • Coenzyme Q10, evidence  Mayo Clinic
  • Hartley R et al, 2013, Green and black tea to prevent cardiovascular disease  Cochrane
  • Sahebkar A et al, 2015, Lack of efficacy of resveratrol on C-reactive protein and selected cardiovascular risk factors–Results from a systematic review and meta-analysis of randomized controlled trials  PubMed
  • Flaxseed oil  WebMD
  • Soy WebMD
  • Vitamin E evidence  Mayo Clinic
  • Myung S-K et al, 2013, Efficacy of vitamin and antioxidant supplements in prevention of cardiovascular disease: systematic review and meta-analysis of randomised controlled trials  The BMJ
  • Choline  WebMD
  • Qidwai W et al, 2013, Role of Garlic Usage in Cardiovascular Disease Prevention: An Evidence-Based Approach  PubMed
  • Genser B et al, 2012, Plant sterols and cardiovascular disease: a systematic review and meta-analysis  PubMed
  • Gupta AK et al, 2011, Role of phytosterols in lipid-lowering: current perspectives  QJM
  • Magnesium  Linus Pauling Institute
  • Calcium intake and risk of cardiovascular disease  PubMed
  • 2012, Potassium intake for adults and children  World Health Organization
  • Siri-Tarino PW et al, 2010, Saturated Fatty Acids and Risk of Coronary Heart Disease: Modulation by Replacement Nutrients  PubMed Central
  • Schwingshackl L et al, 2012, Monounsaturated Fatty Acids and Risk of Cardiovascular Disease: Synopsis of the Evidence Available from Systematic Reviews and Meta-Analyses  PubMed Central
  • Ding EL, Hutfless SM, Ding X, Girota S, 2006, Chocolate and Prevention of Cardiovascular Disease: A Systematic Review  PubMed
  • Khawaja O et al, 2011, Chocolate and coronary heart disease: a systematic review  PubMed
  • Xing X et al, 2014, Herbal Medicines for Cardiovascular Diseases  PubMed Central
  • Luo J et al, Outcome Measures of Chinese Herbal Medicine for Coronary Heart Disease: An Overview of Systematic Reviews  PubMed Central
  • Walden R et al, 2011, Cardiovascular disease Herbal Medicine: Biomolecular and Clinical Aspects, 2nd edition
  • Omega-3 supplements: in depth  National Center for Complementary and Integrative Health
  • Kris-Etherton PM et al, 2002, Fish Consumption, Fish Oil, Omega-3 Fatty Acids, and Cardiovascular Disease  Circulation
  • Aggarwal BB et al, 2008, Potential Therapeutic Effects of Curcumin, the Anti-inflammatory Agent, Against Neurodegenerative, Cardiovascular, Pulmonary, Metabolic, Autoimmune and Neoplastic Diseases  PubMed
  • Bordia A et al, 1997, Effect of ginger (Zingiber officinale Rosc.) and fenugreek (Trigonella foenumgraecum L.) on blood lipids, blood sugar and platelet aggregation in patients with coronary artery disease  PubMed
  • Key TJ et al, 1999, Mortality in vegetarians and nonvegetarians: detailed findings from a collaborative analysis of 5 prospective studies  The American Journal of Clinical Nutrition
  • Dinu M et al, 2016, Vegetarian, vegan diets and multiple health outcomes: a systematic review with meta-analysis of observational studies  PubMed
  • Huang T et al, 2012, Cardiovascular disease mortality and cancer incidence in vegetarians: a meta-analysis and systematic review  PubMed
  • Buckland G et al, 2009, Adherence to the Mediterranean diet and risk of coronary heart disease in the Spanish EPIC Cohort Study  PubMed
  • Martínez-González MA et al, 2011, Mediterranean diet and the incidence of cardiovascular disease: a Spanish cohort  PubMed
  • Mente A et al, 2009, A systematic review of the evidence supporting a causal link between dietary factors and coronary heart disease  PubMed
  • Fidanza F et al, 2004, Mediterranean Adequacy Index: correlation with 25-year mortality from coronary heart disease in the Seven Countries Study  PubMed
  • Panagiotakos DB et al, 2004, Can a Mediterranean diet moderate the development and clinical progression of coronary heart disease? A systematic review.  PubMed
  • Gupta SC et al, 2013, Therapeutic Roles of Curcumin: Lessons Learned from Clinical Trials  PubMed Central
  • Campbell F et al, 2013, A systematic review of fish-oil supplements for the prevention and treatment of hypertension  PubMed
  • Mozaffarian D et al, 2006, Trans Fatty Acids and Cardiovascular Disease  The New England Journal of Medicine
  • Teegala SM et al, 2009, Consumption and health effects of trans fatty acids: a review  PubMed
  • Mente A et al, 2009, A systematic review of the evidence supporting a causal link between dietary factors and coronary heart disease  PubMed
  • Ascherio A et al, 1999, Trans Fatty Acids and Coronary Heart Disease  The New England Journal of Medicine
  • 2007, Opinion of the Scientific Panel on Dietetic products, nutrition and allergies [NDA] related to the presence of trans fatty acids in foods and the effect on human health of the consumption of trans fatty acids  European Food Safety Authority
  • Chowdhury R et al, 2012, Association between fish consumption, long chain omega 3 fatty acids, and risk of cerebrovascular disease: systematic review and meta-analysis  The BMJ
  • Mohebi-Nejad A et al, 2014, Omega-3 Supplements and Cardiovascular Diseases  PubMed Central
  • Aburto NJ et al, 2013, Effect of lower sodium intake on health: systematic review and meta-analyses  The BMJ
  • He FJ et al, 2013, Effect of longer term modest salt reduction on blood pressure: Cochrane systematic review and meta-analysis of randomised trials  The BMJ
  • Micha R et al, 2010, Red and processed meat consumption and risk of incident coronary heart disease, stroke, and diabetes mellitus: a systematic review and meta-analysis  PubMed Central
  • Lam YJT, Atherosclerosis  Merck Manual Professional Edition
  • Kälsch P et al, 2014, Are air pollution and traffic noise independently associated with atherosclerosis: the Heinz Nixdorf Recall Study  PubMed
  • Villines TC et al, 2012, Niacin: the evidence, clinical use, and future directions  PubMed
  • Ruparelia N et al, 2011, Effects of niacin on atherosclerosis and vascular function PubMed Central
  • Alaeddini J, Angina pectoris treatment & management  Emedicine
  • Siphai I et al, 2007, Beta-blockers and progression of coronary atherosclerosis: pooled analysis of 4 intravascular ultrasonography trials  PubMed
  • Kalanuria AA et al, 2012, The prevention and regression of atherosclerotic plaques: emerging treatments  PubMed Central
  • 2008, Aggressively Lowering Cholesterol and Blood Pressure May Reverse Atherosclerosis in Adults with Diabetes  National Institute of Health
  • Centers for Disease Control and Prevention (US); National Center for Chronic Disease Prevention and Health Promotion (US); Office on Smoking and Health (US), 2010, Cardiovascular diseases  How Tobacco Smoke Causes Disease: The Biology and Behavioral Basis for Smoking-Attributable Disease: A Report of the Surgeon General
  • Shai I et al, 2010, Dietary intervention to reverse carotid atherosclerosis  PubMed
  • Hooper L et al, 2015, Effect of cutting down on the saturated fat we eat on our risk of heart disease  Cochrane
  • Mozaffarian D et al, 2010, Effects on coronary heart disease of increasing polyunsaturated fat in place of saturated fat: a systematic review and meta-analysis of randomized controlled trials  PubMed
  • Jakobsen MU et al, 2009, Major types of dietary fat and risk of coronary heart disease: A pooled analysis of 11 cohort studies  NutritionEvidenceLibrary.gov
  • Santanam N et al, 2012, Nicotinic acetylcholine receptor signaling in atherogenesis  PubMed

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